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nani: 28 Sep : 04:31 AM

plz pleasec tell me where to do phd in india

Nikhilphysio: 02 Jun : 03:55 AM

I am working as physiotherapist in Shalby hospital ahmedabad for 4 years. I have passed out from Rajiv gandhi university of health and sciences Bangalore. I want to apply for Newzealand physiotherapy board registration so anyone there from India who got registered as physiotherapist in new zealand please help me.

Arun: 10 May : 12:36 AM

Hi Priyank, welcome. Feel free to go through these forum threads returned by search [link]

Priyank: 09 May : 10:28 PM

Hi..need advice. What are the options in Australia after MPT?

Arun: 04 Mar : 02:01 AM

Happy birthday Boopathi and somasimple


Physio Blogs by Robin

Jan 01 2011

My revision on Chronic Obstructive Pulmonary Disease and Physical Therapy.

COPD is defined as the disease state characterised by limitation in airflow which is progressive and not completely reversible.
Physiological terms associated with the same are:
  • Ventilation: Movement of air thru conducting pathways
  • Respiration: exchange of gases inside the body
  • Tidal volume: amount of air inspired and expired during normal ventilation
  • Total lung capacity: the amount of air contained in the lungs after a forced inspiration
  • Inspiratory reserve volume: amount of air forcefully inspired after a normal inspiration
  • Expiratory reserve volume: amount of air expired forcefully after the normal expiration
  • Residual volume: amount of air left in the lungs even after a forced expiration
  • Vital capacity: amount of air that is moved during forced inspiration and forced expiration.
The GOLD (Global initiative for chronic Obstructive Lung Disease) classification is:
  • 0 : at risk
  • 1 : mild
  • 2 : moderate
  • 3 : severe
  • 4 : very severe

I was with the Pathophysiology today and these were the points i noted:
  • COPD is a combination of airway narrowing, parenchymal destruction and pulmonary vascular thickening which is due to chronic airway inflammation.
  • More pronounced in peripheral pathways.
  • Chronic inflammation leads to increased neutrophils , macrophages and Tlymphocytes in the area causing endothelial damages.
  • Damage leads to repair and remodelling of the pathways.
  • Remodelled pathways has globlet cells and glands which are hypertrophied producing excess secretion.
  • Coupled with decreased ciliary function it impairs airway clearance.
  • Damaged and irritated mucosa also is increased in sensitivity to irritants leading to bronchial hyperactivity.
  • Dialation and destruction of respiratory bronchioles.
  • Imbalances in protienase and antiproteinase leads to loss in elastic recoil properties of lungs.
  • Destruction of pulmonary capillary bed in advanced stages.
  • Ventilation/perfusion ratio shows great mismatch leading to Hypoxemia.
  • Hypoxemia leads to hypercapnea in later stages.
  • Capillary wall destruction leads to increased pulmonary vascular resistance which inturn leads to right ventricular hypertrophy termed 'Cor Pulmonale'
  • Polycythemia in advanced stages.